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High Quality Huperzine-a on Sell
| Price | $10.00 |
|---|---|
| Min Order | 1 kg |
| Availability | In Stock |
| Shipping From | Jiangsu, China |
| Popularity | 383 people viewed |
Model NO.:
GRJ20150314
Certification:
ISO
Assay Method:
HPLC
Application Form:
Tablet, Capsule
State:
Powder
Extract Source:
Huperzine-a
Odor:
Characteristic
Delivery:
in Time
Taste:
Characteristic
Test Method:
HPLC
Trademark:
Gorunjie
Transport Package:
25kg/Drum or at Your Requirements
Specification:
ISO
Origin:
China
HS Code:
2932999099
Product Description
We provide Huperzine-A from Huperzia serrta.
Assay: 98%Huperzine-A
Molecular Formula: C<sub>15</sub>H<sub>18</sub>N<sub>2</sub>O
Molecular Weight: 242.32
CAS Registry Number: 102518-79-6
Huperzine A is a plant alkaloid derived from the Chinese club moss plant, Huperzia serrata, which is a member of the Lycopodium species. Huperzia serrata has been used in Chinese folk medicine for the treatment of fevers and inflammation.
Huperzine A has been found to have acetylcholinesterase activity. In Chinese medicine, the extract of Huperzia serrata is known as Chien Tseng Ta and shuangyiping. Huperzine A derivatives are being developed for pharmaceutical application.
Huperzine A may have cognition-enhancing activity, can maintains acetylcholine levels for healthy cognitive function and promotes enhanced memory and cell to cell communication.
[Description]
Huperzine A has been proven superior to drugs licensed for the treatment of Alzheimer's, including the leading anticholine sterase drugs physostigmine and tacrine. Huperzine A improved learning and memory in mice better than tacrine. Unlike physostigmine and tacrine, Huperzine A acts specifically on AchE in the brain rather than AchE found elsewhere in the body i.e., far less is wasted on irrelevant effects. Unlike physostigmine and tacrine, Huperzine A does not appear to bind to receptors in the central nervous system, which can cause adverse side effects. Its effects last 10 to 12 times longer than physostigmine and tacrine (up to 8 hours).
[Main Function]
1)huperzine A can improve memory, thinking, and behavioral function in people with
Alzheimer's disease, multi-infarct dementia, and senile dementia, and increase
the level of the neurotransmitter acetylcholine by blocking its breakdown.
2) It may also protect neurons from cell death caused by toxic levels of glutamate
and protect against some of the effects of chemical nerve agents used in warfare,
.
3) It is also the treatment of AD in a randomized controlled trial of its effect
on cognitive function.
Assay: 98%Huperzine-A
Molecular Formula: C<sub>15</sub>H<sub>18</sub>N<sub>2</sub>O
Molecular Weight: 242.32
CAS Registry Number: 102518-79-6
Huperzine A is a plant alkaloid derived from the Chinese club moss plant, Huperzia serrata, which is a member of the Lycopodium species. Huperzia serrata has been used in Chinese folk medicine for the treatment of fevers and inflammation.
Huperzine A has been found to have acetylcholinesterase activity. In Chinese medicine, the extract of Huperzia serrata is known as Chien Tseng Ta and shuangyiping. Huperzine A derivatives are being developed for pharmaceutical application.
Huperzine A may have cognition-enhancing activity, can maintains acetylcholine levels for healthy cognitive function and promotes enhanced memory and cell to cell communication.
[Description]
Huperzine A has been proven superior to drugs licensed for the treatment of Alzheimer's, including the leading anticholine sterase drugs physostigmine and tacrine. Huperzine A improved learning and memory in mice better than tacrine. Unlike physostigmine and tacrine, Huperzine A acts specifically on AchE in the brain rather than AchE found elsewhere in the body i.e., far less is wasted on irrelevant effects. Unlike physostigmine and tacrine, Huperzine A does not appear to bind to receptors in the central nervous system, which can cause adverse side effects. Its effects last 10 to 12 times longer than physostigmine and tacrine (up to 8 hours).
[Main Function]
1)huperzine A can improve memory, thinking, and behavioral function in people with
Alzheimer's disease, multi-infarct dementia, and senile dementia, and increase
the level of the neurotransmitter acetylcholine by blocking its breakdown.
2) It may also protect neurons from cell death caused by toxic levels of glutamate
and protect against some of the effects of chemical nerve agents used in warfare,
.
3) It is also the treatment of AD in a randomized controlled trial of its effect
on cognitive function.
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