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Purity Nootropic Tianeptine Sodium Anti-Depression Tianeptine Sodium Raw Powder Tianeptine Sodium

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Xi`an Disheng Health Biotechnology Co., Ltd.

Audited Supplier 5 years

Profile Certified by SGS/BV

Shaanxi, China
Victor Chen (Mr.)

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Model NO.:

D-Sung Tianeptine Sodium

Powder:

Yes

Customized:

Customized

Certification:

GMP, HSE, ISO 9001, USP, BP

Suitable for:

Elderly, Adult

State:

Powder

Purity:

>99%

Product Name:

Tianeptine Sodium

Name:

Tianeptine Sodium Powder

Appearance:

White Powder

Color:

White

Store:

Cool and Dry

Grade:

Pharmaceutical Grade

Assay:

HPLC 99%

Application:

Anthelmintics

Specific:

COA

Test Method:

HPLC

Shelf Life:

2 Years

CAS No.:

30123-17-2

Mf:

C21h24cln2nao4s

Density:

1.38

Melting Point:

1800 C

Trademark:

D-Sung

Transport Package:

Negotiable

Specification:

99%

Origin:

Xi'an Shaanxi

Product Description

Purity Nootropic Tianeptine Sodium Anti-Depression Tianeptine Sodium Raw Powder Tianeptine Sodium

Product name	Tianeptine Sodium
Appearance	White powder
CAS	30123-17-2
MF	C21h24cln2nao4s

Tianeptine sodium has structural similarities to the tricyclic antidepressants, but it has different pharmacological properties. Tianeptine is a selective serotonin reuptake enhancer (SSRE), opposite to the action of SSRIs. One review points to the cancellative effects of tianeptine and fluoxetine coadministration on serotonin reuptake. Another suggests that long-term administration of tianeptine has no effect on serotonin pathways. Tianeptine enhances the extracellular concentration of dopamine in the nucleus accumbens and modulates the D2 and D3 dopamine receptors. There is also action on the NMDA and AMPA receptors.

Tianeptine sodium is a drug used primarily in the treatment of major depressive disorder, although it may also be used to treat asthma or irritable bowel syndrome. Chemically it is a tricyclic antidepressant (TCA), but it has different pharmacological properties than typical TCAs as recent research suggests that tianeptine produces its antidepressant effects through indirect alteration of glutamate receptor activity (i.e., AMPA receptors and NMDA receptors) and release of BDNF, in turn affecting neural plasticity.